Progeria
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Lysosome activation clears progerin and eases cellular aging in progeria, study finds
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Scientists in China report that repairing defects in lysosomes—the cell’s waste‑disposal hubs—accelerated clearance of progerin in patient cells and reduced markers of cellular aging, pointing to a potential therapeutic target for Hutchinson‑Gilford progeria syndrome.
Researchers report that a variant of the BPIFB4 gene enriched in long‑lived people improved markers of heart health in mouse and cell models of Hutchinson–Gilford progeria syndrome, suggesting a potential new path for therapy.