Scientists at Stanford University have discovered that hyperactivity in the brain's reticular thalamic nucleus may cause autism-like behaviors in mice. By suppressing this overactivity with drugs or neuromodulation, symptoms were reversed in mouse models. This finding suggests potential new therapeutic approaches overlapping with epilepsy treatments.

Key Details

Researchers identified the reticular thalamic nucleus as a key area driving autism symptoms. In experiments, targeted interventions normalized behaviors in mice, offering hope for human applications.

Implications

The study, published on September 8, 2025, highlights shared pathways with epilepsy, potentially accelerating drug development. Further human trials are needed.

Source: California Healthline