Cuba launches clinic for chikungunya aftereffects

The Institute of Neurology and Neurosurgery in Cuba has begun a specialized clinic to care for patients with post-chikungunya ailments, particularly those linked to neuropathic pain, joint pain, and paresthesia. This step provides focused treatment for individuals impacted by the mosquito-borne virus.

In Havana on December 23, the Institute of Neurology and Neurosurgery in Cuba has launched a specialized clinic dedicated to treating patients with post-chikungunya ailments. The facility focuses on conditions such as neuropathic pain, joint pain, and paresthesia, which are common lingering effects of the mosquito-borne chikungunya virus that causes fever and severe aches.

Chikungunya can lead to long-term complications that impair daily life for those infected. The new clinic aims to address these persistent issues through comprehensive care. As reported by Prensa Latina, this opening meets the demand for specialized health services in Cuba.

While specific patient numbers are not yet available, the clinic is part of Cuba's public health efforts to manage infectious diseases and their neurological aftermaths. This initiative highlights Cuba's dedication to accessible medical treatment for virus-related conditions.

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Illustration of mitochondria transferring from glia to neurons to reduce nerve pain in neuropathy models.
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Duke-led Nature study links glia-to-neuron mitochondria transfer to reduced nerve pain in neuropathy models

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Duke University researchers report that boosting the transfer of healthy mitochondria from support cells to sensory neurons reduced pain-like behaviors in mouse models of diabetic and chemotherapy-related peripheral neuropathy, an approach they say could address a root driver of nerve pain rather than simply blocking pain signals.

Researchers at the University of Colorado Boulder have pinpointed a brain region called the caudal granular insular cortex, or CGIC, that acts as a switch turning acute pain into chronic pain. In animal studies, disabling this circuit prevented chronic pain from developing or reversed it once established. The findings, published in the Journal of Neuroscience, open paths to new treatments beyond opioids.

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