Medical illustration showing Enterococcus faecalis bacteria producing hydrogen peroxide to stall chronic wound healing, with catalase enzyme restoring skin cell migration.
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Study links Enterococcus faecalis metabolism to stalled healing in chronic wounds

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Researchers report that Enterococcus faecalis—a bacterium often found in chronic wounds—can hinder skin repair by generating hydrogen peroxide through a metabolic pathway, triggering stress responses that stop key skin cells from migrating. In laboratory experiments, breaking down the peroxide with the antioxidant enzyme catalase helped restore cell movement, suggesting a potential treatment approach that does not rely on antibiotics.

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