Studi Cambridge ungkap potensi untuk memulihkan kerusakan saraf

Para peneliti di University of Cambridge telah mengembangkan model laboratorium miniatur otak dan sumsum tulang belakang manusia yang menunjukkan bahwa serabut saraf yang rusak dapat memperoleh kembali kemampuan untuk tumbuh kembali di bawah kondisi tertentu.

Tim tersebut membangun sirkuit saraf fungsional menggunakan organoid yang berasal dari sel punca yang dijaga agar tetap terpisah secara fisik di laboratorium. Akson dari jaringan otak tumbuh melintasi celah tersebut untuk terhubung dengan jaringan sumsum tulang belakang dan memicu kontraksi otot. Pengaturan ini memungkinkan studi mengenai regenerasi selama lebih dari setahun masa perkembangan.

Artikel Terkait

Illustration of mitochondria transferring from glia to neurons to reduce nerve pain in neuropathy models.
Gambar dihasilkan oleh AI

Duke-led Nature study links glia-to-neuron mitochondria transfer to reduced nerve pain in neuropathy models

Dilaporkan oleh AI Gambar dihasilkan oleh AI Fakta terverifikasi

Duke University researchers report that boosting the transfer of healthy mitochondria from support cells to sensory neurons reduced pain-like behaviors in mouse models of diabetic and chemotherapy-related peripheral neuropathy, an approach they say could address a root driver of nerve pain rather than simply blocking pain signals.

Researchers in Japan have created new vitamin K compounds that are three times more effective than natural forms at turning stem cells into neurons. The work, published in 2025, targets diseases that destroy brain cells such as Alzheimer’s and Parkinson’s.

Dilaporkan oleh AI

Researchers have shown that rejuvenating muscle stem cells from old mice leads to larger muscle growth and improved recovery after injury. The approach could one day help older people regain strength and mobility. Experiments demonstrated clear benefits in leg function and tissue repair.

A widely studied anti-aging treatment triggered significant brain damage in mice, according to new research from the University of Connecticut. The drug combination dasatinib plus quercetin caused myelin loss and changes resembling those seen in multiple sclerosis. The findings raise questions about its use in longevity studies and off-label therapies.

Situs web ini menggunakan cookie

Kami menggunakan cookie untuk analisis guna meningkatkan situs kami. Baca kebijakan privasi kami untuk informasi lebih lanjut.
Tolak